Genetics & Heredity |
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Johann Gregor Mendel (1822-1884)
The Man, the Monk Johann Gregor Mendel was born in the Silesian village of Heinzendorf, now Hyncice in the Czech Republic. His parents were peasant farmers and very early on recognized their son's intellect. Mendel was able to stay in school and pursue an academic life. His sister, Theresia, actually sacrificed part of her dowry so that Mendel could get an education. In 1843, Mendel entered the Augustinian Monastery in Brno(in what is now the Czech Republic) as a novice. In his autobiography, Mendel said that unlike other clerics, he didn't feel called to the Church: "my circumstances decided my vocational choice." ...Read More |
Interactive Science Notebook |
Quizlet Reviews |
What is DNA? Notes
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Shape of DNA
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DNA to the Cell
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DNA Foldable
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Unit 1 - DNA/RNA Notes Click on the above link and record your DNA/RNA Notes in your Interactive Science Notebook Explore: DNA Extraction LabOnline Resources |
Activity: Codon Table
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What is the DNA Structure?
BrainPop Video: DNA
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Nucleic Acids: DNA and RNA
DNA Replication: Copying the Molecule of Life
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DNA Replications
DNA, Chromosomes, Genes & Traits
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Genetic Mutations: Tay-Sach's Disease
Nathan's Story
Understanding Tay-Sachs Disease
In 2011, our son Nathan Harney is almost 17 months old. He is a wonderful, happy baby who has beautiful blue eyes, a huge personality, and is the light of our lives. He is a gentle soul that we have been blessed with. He was initially diagnosed with a developmental delay somewhere around 10 months old. He does not walk, he no longer stands or rolls over, and can no longer sit unassisted. He does, however, yell “up” when he wants to be picked up, wiggles like crazy when his daddy sings to him, yells “whoa” every morning in his crib when he wakes up, and still says “oh ma ma” when he cries. He has a huge heart for such a little guy, and his smile can light up a room.We began to notice at an early age that he was losing skills... Read More
Understanding Tay-Sachs Disease
In 2011, our son Nathan Harney is almost 17 months old. He is a wonderful, happy baby who has beautiful blue eyes, a huge personality, and is the light of our lives. He is a gentle soul that we have been blessed with. He was initially diagnosed with a developmental delay somewhere around 10 months old. He does not walk, he no longer stands or rolls over, and can no longer sit unassisted. He does, however, yell “up” when he wants to be picked up, wiggles like crazy when his daddy sings to him, yells “whoa” every morning in his crib when he wakes up, and still says “oh ma ma” when he cries. He has a huge heart for such a little guy, and his smile can light up a room.We began to notice at an early age that he was losing skills... Read More
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What is a Genetic Mutation?
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Mutations in DNA
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DNA Structure & Replication
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Interactive Science Notebook: Protein Synthesis
Mutation Labs/Activities
How DNA Determines Traits-Unigriffins
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Mutations Foldable
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Sickle Cell Anemia
Sickle cell anemia is an inherited form of anemia — a condition in which there aren't enough healthy red blood cells to carry adequate oxygen throughout your body.
Normally, your red blood cells are flexible and round, moving easily through your blood vessels. In sickle cell anemia, the red blood cells become rigid and sticky and are shaped like sickles or crescent moons. These irregularly shaped cells can get stuck in small blood vessels, which can slow or block blood flow and oxygen to parts of the body. There's no cure for most people with sickle cell anemia. But treatments can relieve pain and help prevent problems associated with the disease. Sickle cell anemia is caused by a mutation in the gene that tells your body to make the red, iron-rich compound that gives blood its red color (hemoglobin). Hemoglobin allows red blood cells to carry oxygen from your lungs to all parts of your body. In sickle cell anemia, the abnormal hemoglobin causes red blood cells to become rigid, sticky and misshapen. The sickle cell gene is passed from generation to generation in a pattern of inheritance called autosomal recessive inheritance. This means that both the mother and the father must pass on the defective form of the gene for a child to be affected...Read More Click here to see 3D Animation of Sickle Cell Anemia |
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